Delayed-Onset Striatal Hands Following Basal Ganglia Anoxic Insult
نویسندگان
چکیده
منابع مشابه
Striatal inhomogeneities and basal ganglia function.
It has always been something of a mystery as to why different diseases of the basal ganglia may produce completely opposite effects on movement. Thus, defective dopaminergic inhibitory input into the striatum in Parkinson’s disease causes an akinetic-rigid syndrome, while destruction of the striatum in Huntington’s disease usually causes chorea. This theoretic paradox may be resolved by more de...
متن کاملStriatal Plasticity and Basal Ganglia Circuit Function
The dorsal striatum, which consists of the caudate and putamen, is the gateway to the basal ganglia. It receives convergent excitatory afferents from cortex and thalamus and forms the origin of the direct and indirect pathways, which are distinct basal ganglia circuits involved in motor control. It is also a major site of activity-dependent synaptic plasticity. Striatal plasticity alters the tr...
متن کاملCentrality of Striatal Cholinergic Transmission in Basal Ganglia Function
Work over the past two decades revealed a previously unexpected role for striatal cholinergic interneurons in the context of basal ganglia function. The recognition that these interneurons are essential in synaptic plasticity and motor learning represents a significant step ahead in deciphering how the striatum processes cortical inputs, and why pathological circumstances cause motor dysfunctio...
متن کاملDysprosodic speech following basal ganglia insult: toward a conceptual framework for the study of the cerebral representation of prosody.
Progress in understanding brain/behavior relationships in adult-acquired dysprosody has led to models of cortical hemispheric representation of prosodic processing based on functional (linguistic vs affective) or physical (timing vs pitch) parameters. These explanatory perspectives have not been reconciled, and also a number of neurobehavior syndromes that include dysprosody among their neurolo...
متن کاملDelayed post-anoxic encephalopathy.
Neurologic deficits following acute anoxic insults are well recognised. However, severe neurologic dysfunction may be a delayed postanoxic manifestation.'`3 These are of importance due to their rare occurrence, distinctive clinical and pathologic features, and spontaneous resolution in the majority of cases.2 Most of these delayed post-anoxic neurologic sequelae have been described following ca...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Acta Médica Portuguesa
سال: 2017
ISSN: 1646-0758,0870-399X
DOI: 10.20344/amp.7772